Browsing by Author "Hicks, Joanna"
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The inflection point hypothesis: The relationship between the temperature dependence of enzyme-catalyzed reaction rates and microbial growth rates.
(2020)The temperature dependence of biological rates at different scales (from individual enzymes to isolated organisms to ecosystem processes such as soil respiration and photosynthesis) is the subject of much historical and ...
Co-authors for Joanna Hicks
Supervised by Joanna Hicks
Showing up to 5 theses - most recently added to Research Commons first.
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Alternative sulfur acquisition pathways in Neisseria gonorrhoeae
(The University of Waikato, 2021)Neisseria gonorrhoeae is an obligate human pathogen responsible for the sexually transmitted infection, gonorrhoea. Its success as a pathogen is partly due to robust defence mechanisms that provide protection against ... -
Structural and biochemical characterisation of O-acetylserine sulphydrylase (CysK) from Neisseria gonorrhoeae
(The University of Waikato, 2021)Neisseria gonorrhoeae is the causative bacteria of the sexually transmitted infection (STI), gonorrhoea. Rapid emergence of antibiotic resistant strains of N. gonorrhoeae has given rise to almost untreatable gonorrhoea ... -
DNA repair during bacterial competence? A potential role for the periplasmic ligase, Lig E
(The University of Waikato, 2021)The rapid rise in antibiotic resistance among pathogenic bacteria calls for better understanding of drivers of resistance and pathways by which it occurs. Identification of such mechanisms could identify novel pathways ... -
Cysteine biosynthesis and the role of CysE in Neisseria gonorrhoeae
(The University of Waikato, 2020)Neisseria gonorrhoeae is the causative organism of the sexually transmitted infection (STI), gonorrhoea. Over decades of antibiotic use, the emergence of antibiotic-resistant strains of N. gonorrhoeae has dramatically ... -
Characterisation of the FitAB Toxin-Antitoxin System in Neisseria gonorrhoeae
(The University of Waikato, 2019)Neisseria gonorrhoeae is the causative agent of the sexually transmitted infection, gonorrhoea. In a standard infection, N. gonorrhoeae invade epithelial cells lining the urogenital tract and exit into the sub-epithelial ...
