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dc.contributor.authorJacobson, Gregory M.
dc.contributor.authorVoss, Logan J.
dc.contributor.authorMelin, Sofia M.
dc.contributor.authorMason, Jonathan P.
dc.contributor.authorCursons, Raymond T.
dc.contributor.authorSteyn-Ross, D. Alistair
dc.contributor.authorSteyn-Ross, Moira L.
dc.contributor.authorSleigh, James W.
dc.coverage.spatialNetherlandsen_NZ
dc.date.accessioned2010-09-28T23:26:06Z
dc.date.available2010-09-28T23:26:06Z
dc.date.issued2010
dc.identifier.citationJacobson, G.M., Voss, L.J., Melin, S.M., Mason, J.P., Cursons, R.T.,…, Sleigh, J.W. (2010). Connexin36 knockout mice display increased sensitivity to pentylenetetrazol-induced seizure-like behaviours. Brain Research, 1360(11), 198-204.en_NZ
dc.identifier.urihttps://hdl.handle.net/10289/4613
dc.description.abstractLarge-scale synchronous firing of neurons during seizures is modulated by electrotonic coupling between neurons via gap junctions. To explore roles for connexin36 (Cx36) gap junctions in seizures, we examined the seizure threshold of connexin36 knockout (Cx36KO) mice using a pentylenetetrazol (PTZ) model.en_NZ
dc.format.mimetypeapplication/pdf
dc.language.isoen
dc.publisherElsevieren_NZ
dc.rightsThis is an author’s accepted version of an article published in the journal: Brain Research. © 2010 Elsevier.en_NZ
dc.subjectEpilepsyen_NZ
dc.subjectCx36en_NZ
dc.subjectgap junctionen_NZ
dc.subjectPTZen_NZ
dc.subjectinterneuronen_NZ
dc.titleConnexin36 knockout mice display increased sensitivity to pentylenetetrazol-induced seizure-like behaviorsen_NZ
dc.typeJournal Articleen_NZ
dc.identifier.doi10.1016/j.brainres.2010.09.006en_NZ
dc.relation.isPartOfBrain Researchen_NZ
pubs.begin-page198en_NZ
pubs.elements-id4705
pubs.end-page204en_NZ
pubs.issue11en_NZ
pubs.volume1360en_NZ
uow.identifier.article-noCen_NZ


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